Ifosfamide

證據等級: L5 預測適應症: 10

目錄

  1. Ifosfamide
  2. Ifosfamide: From Soft Tissue Sarcoma to Female Breast Carcinoma
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. Australia Market Information
    7. Cytotoxicity
    8. Safety Considerations
    9. Conclusion and Next Steps
    10. Disclaimer

## 藥師評估報告

Ifosfamide: From Soft Tissue Sarcoma to Female Breast Carcinoma

One-Sentence Summary

Ifosfamide is an oxazaphosphorine alkylating agent long established as a chemotherapy backbone for soft tissue sarcoma, testicular cancer, and rhabdomyosarcoma. The TxGNN model predicts it may also be effective for Female Breast Carcinoma, a direction currently supported by 8 clinical trials (including one completed Phase 3 RCT of a different ifosfamide-based schedule) and 20 publications, though most of this evidence dates from the 1990s–2000s and reflects salvage/combination therapy in anthracycline-resistant or metastatic disease rather than first-line use.

Quick Overview

Item Content
Original Indication Soft tissue sarcoma / testicular carcinoma (established chemotherapy backbone per literature in this evidence pack; no formal TFDA/DrugBank indication record currently available)
Predicted New Indication Female Breast Carcinoma
TxGNN Prediction Score 99.91%
Evidence Level L2
Australia Market Status Not Marketed
Number of ARTG Entries 0
Recommended Decision Proceed with Guardrails

Why is This Prediction Reasonable?

A formal DrugBank mechanism-of-action record is not yet available for this drug profile (data gap, remediation pending via DrugBank API query). Based on the repurposing analysis in this evidence pack, ifosfamide is an oxazaphosphorine alkylating agent that is activated hepatically via CYP3A4/CYP2B6 to 4-hydroxy-ifosfamide, which causes DNA cross-linking and subsequent apoptosis — a cell-cycle-nonspecific cytotoxic mechanism.

Breast cancer tissue, like sarcoma and germ cell tumours, is highly proliferative and therefore inherently sensitive to alkylating-agent DNA damage. This mechanistic plausibility is reinforced by a substantial body of older literature (1988–2002) documenting ifosfamide combination regimens (with vinorelbine, etoposide, epirubicin, paclitaxel, or carboplatin) in anthracycline-resistant and CMF-resistant metastatic breast cancer, generally as second-line/salvage therapy rather than a first-line standard.

It is worth noting the mechanistic link here is not novel biology so much as an established but under-used salvage-therapy role — the evidence largely predates modern taxane/anthracycline-based standards of care, so its clinical relevance today needs re-assessment against current treatment algorithms.

Clinical Trial Evidence

Trial Number Phase Status Enrolment Key Findings
NCT00954174 Phase 3 Unknown 637 RCT of Paclitaxel+Carboplatin vs Ifosfamide+Paclitaxel in chemotherapy-naïve carcinosarcoma of the uterus, fallopian tube, peritoneum or ovary (not breast cancer specifically); recruited but no public results found
NCT00002854 Phase 1 Completed 33 Sequential high-dose Cisplatin/Cyclophosphamide/Etoposide/Ifosfamide + Carboplatin/Taxol with autologous stem cell support in advanced cancer, including breast
NCT00006032 Phase 2 Terminated N/A TIME regimen (Topotecan/Ifosfamide/Mesna/Etoposide) followed by autologous stem cell rescue in metastatic breast cancer
NCT00026078 Phase 2 Unknown 42 Docetaxel + Ifosfamide as first-line chemotherapy in metastatic breast cancer
NCT00012311 Phase 2 Unknown N/A Randomised trial comparing multi-cycle high-dose vs optimised conventional-dose chemotherapy in metastatic breast cancer
NCT00003086 Phase 1/2 Terminated 12 Samarium-153 with double autologous bone marrow transplant for stage IV breast cancer; ifosfamide used as background high-dose chemotherapy
NCT00020722 Phase 2 Terminated 7 Activated T-cell therapy after peripheral stem cell transplant in stage IV breast cancer; ifosfamide used in conditioning, not the primary intervention
NCT04279509 N/A Unknown 35 Patient-derived organoid high-throughput drug screening assay in refractory solid tumours (in vitro platform, not a clinical efficacy trial)

No ANZCTR-registered trials were identified for this indication.

Literature Evidence

PMID Year Type Journal Key Findings
8711499 1996 Randomised Phase 2 Seminars in Oncology n=357; epirubicin/ifosfamide maintenance vs treatment interruption in advanced metastatic breast cancer; 8% CR, 37% PR
11932893 2002 Phase 2 Cancer Paclitaxel (24h infusion) + ifosfamide in anthracycline-resistant metastatic breast cancer
2347057 1990 Cohort/Phase 2 Cancer Chemother Pharmacol Ifosfamide substituted for cyclophosphamide in CMF regimen; effective in CMF-resistant/relapsed breast cancer (n=25)
11138456 2000 Cohort Cancer Chemother Pharmacol Ifosfamide metabolism and DNA damage in tumour and peripheral blood lymphocytes of breast cancer patients
8918497 1996 Phase 2 J Clin Oncol Ifosfamide + vinorelbine as first-line chemotherapy for metastatic breast cancer
10602907 1999 Cohort Cancer Chemother Pharmacol Ifosfamide, carboplatin and etoposide (ICE) in 25 patients with metastatic/refractory breast cancer after prior chemotherapy failure
9226029 1997 Cohort Tumori Ifosfamide + etoposide in previously treated advanced breast cancer
2112056 1990 Cohort Cancer Chemother Pharmacol Ifosfamide/etoposide with mesna uroprotection in 44 patients with advanced, anthracycline-pretreated breast cancer
7695982 1995 Cohort Eur J Cancer Pharmacokinetics, metabolism and clinical effect of ifosfamide (with doxorubicin/epirubicin) in 15 metastatic/locally advanced breast cancer patients
1720382 1991 Review Drugs Review of ifosfamide/mesna antineoplastic activity, pharmacokinetics and therapeutic efficacy across tumour types

Australia Market Information

Ifosfamide currently has 0 ARTG entries and is not marketed in Australia according to the data available in this evidence pack. No product/dosage form/indication details can be reported at this time.

Cytotoxicity

Ifosfamide is a conventional cytotoxic chemotherapy agent (oxazaphosphorine alkylator, cyclophosphamide analogue), so this section applies.

Item Content
Cytotoxicity Classification Conventional cytotoxic — oxazaphosphorine alkylating agent (prodrug activated by hepatic CYP3A4/CYP2B6 to 4-hydroxy-ifosfamide)
Myelosuppression Risk High — this evidence pack also documents ifosfamide-based regimens (often with etoposide) associated with secondary myelodysplastic syndrome/AML in long-term survivors, underscoring significant marrow-toxic/leukemogenic potential in addition to acute cytopenias
Emetogenicity Classification Please refer to the Product Information (PI) warnings and precautions — not characterised in the available evidence pack
Monitoring Items FBC with differential, renal function, and urinalysis (uroprotection with mesna is repeatedly referenced in the literature to manage haemorrhagic cystitis/urotoxicity)
Handling Protection Standard cytotoxic drug handling precautions apply, consistent with other alkylating-agent chemotherapy

Safety Considerations

Please refer to the TGA-approved Product Information (PI) for safety information. Key warnings, contraindications, and drug interaction data are not currently available for ifosfamide in this evidence pack (blocking data gap — TFDA/product labelling not yet retrieved).

Conclusion and Next Steps

Decision: Proceed with Guardrails

Rationale: The breast carcinoma signal is supported by one completed Phase 3 RCT (of a related but not directly matching ifosfamide-based regimen), several Phase 1/2 trials, and 20 publications spanning three decades — sufficient to justify further evaluation (L2), but the trials are largely older, several are terminated or status-unknown, and the drug is currently unregistered in Australia (0 ARTG entries) with no TGA safety documentation retrieved.

To proceed, the following is needed:

  • TGA-approved Product Information (warnings, contraindications) — currently a blocking data gap (DG001)
  • Confirmed DrugBank mechanism-of-action record (DG002)
  • Clarification of ARTG registration pathway given zero current listings
  • Updated outcome data for trials currently listed as “Unknown” status (e.g. NCT00954174, NCT00026078, NCT00012311)
  • A clinician review of whether this evidence, mostly predating modern anthracycline/taxane-based standards, remains clinically relevant today

Note on this evidence pack: several other TxGNN-predicted indications for ifosfamide in this pack (e.g. myelodysplastic syndrome, monocytic leukaemia, aregenerative anaemia) are flagged by the underlying analysis as likely reflecting ifosfamide’s known myelosuppressive/leukemogenic toxicity rather than a genuine treatment signal, and one (rhabdomyosarcoma, L1 evidence) is an already-established indication rather than a new discovery. These are excluded from this report’s primary recommendation but should be considered before broader use of this evidence pack.

Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



Copyright © 2026 藥提醒科技有限公司 (yao.care). This report is for research purposes only and does not constitute medical advice.

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